Is cancer caused by negative emotions?

A homeopathic doctor believes that cancer is caused by negative emotions. For instance, cancer in the left breast is born out of a loss, and femininity issues. Cancer in the right breast is spurred by conflicts with the opposite sex. Cancer in the small intestines is a result of prolonged depression. Anger, meanwhile, can result in cancer in the large intestine.

Indian-born Dr. Shaji Kudiyat maintains that the soul or life force, the mind and emotions, and bodily functions are all intimately linked. Positive and negative experiences and impressions can thus have an impact on physical functions. Feelings of anger, worry or sorrow can subsequently make the body more vulnerable.

On the other hand, when the patient awakens its spiritual identity or begins to understands itself as a life force and attunes itself to love and hope, healing can begin.

At the start of his medical practice, Kudiyat realized that cancer was being treated without understanding its root causes. “Today’s cancer treatments mainly focus on the identification and rectification of the tumor growth,” he says. “It is achieved by surgically removing the tumor or by destroying it by chemotherapy or radiation.”

He realize that the things he learned in medical school had their limitations. Science maintains that genes are responsible for life and that the body is made of material components and biochemical actions and reactions. However, they don’t have the ability to think, feel or express emotions.

Based on his researches, Kudiyat says he discovered that there is a nonmaterial element, a living energy, that actually makes the body function.

“This energy can be easily identified as the wave which the doctors and nurses monitor in all intensive care units,” he says. “My curious mind started searching for solutions by which we can overcome scientific limitations.”

‘Dynamic Science’

This led him to form what he calls “Dynamic Science,” which attempts to explain how the mind, the vital force (soul) and DNA (which decides all physical functions) are interconnected.

“It answers many questions and helps solve some difficult problems including diseases like cancer,” says Kudiyat.

“Dynamic Science can explain how our thoughts decide the genetic nature of our own DNA. So when we have positive thoughts, the vibrations can modify the DNA constructively, resulting in a curative DNA.  Similarly, negative thoughts modify the DNA, resulting in the unhealthy transformation of the DNA, producing diseases including cancer.”

Kudiyat observes that cancer patients follow a certain pattern: They held deep-seated negative emotions and hurts before they developed cancer.




“Through Dynamic Science, we identify the root cause of cancer and chronic diseases as  negative mental states other than nutritional deficiency and lack of proper rest, sleep and exercise,” says Kudiyat.

Nonsurgical alternative

He offers a nonsurgical alternative. First, he makes a holistic case study of the patient by knowing the personality. Then the patient and family attend a group counseling session. This includes probing the psychological reasons for the cancer. He  helps the patient understand what is needed to be done to eliminate the causes.

He then prescribes homeopathic medicine and encourages yoga, meditation, prayer, music therapy, laughter therapy and other techniques to let the mind be at peace.

Kudiyat shares a success story. A married lady had conflicts with her husband which remained unresolved for many years. This hardened her. She slowly developed a lump in the right breast which became malignant. After she consulted several doctors, her biopsy confirmed that she had invasive ductal carcinoma.

Although she underwent mastectomy, she also consulted Dr. Kudiyat for a lifestyle change. The patient realized that beneath the hardened heart was a wellspring of peace and happiness. She learned to forgive, let go, move on and trust again.

“That was in 1997; she’s still alive,” he says.

Dr. Shaji Kudiyat will hold a lecture on “Dynamic Science: Approach to Cancer Prevention & Treatment” on Sept. 5, from 6 to 9:30 p.m., at Santuario de San Antonio, Buffalo cor. Duke St., Greenhills. For details, call 0917-5275002.

source: lifestyle.inquirer.net

Cancer gene mutation more complex than previously thought: study

LONDON (Reuters) - Taking a sample or biopsy from just one part of a tumor might not give a full picture of its genetic diversity and may explain why doctors, despite using genetically targeted drugs, are often unable to save patients whose cancer has spread, scientists said.

A study by British researchers found there are more genetic differences than similarities between biopsies taken from separate areas of the same tumor, and yet further gene differences in samples taken from secondary tumors.

That might help explain why, despite recent development of a wave of highly targeted drugs designed to tackle cancers of specific genetic types, the prognosis remains poor for many patients with so-called solid-tumor disease like breast, lung, or kidney cancer that has spread to others parts of the body.

But the researchers, whose study was partly funded by charity Cancer Research UK and published in the New England Journal of Medicine, said it also pointed to a way forward.

The team carried out the first ever genome-wide analysis of the genetic changes or faults in different regions of the same tumor.

They looked at four patients with cancer in their kidneys, taking samples from different regions of the primary tumor and also from other organs where the tumor had spread.

They found that the majority of gene faults, around two-thirds, were not the same in one sample as in another, even when the biopsies were taken from the same tumor.

Samples taken from secondary tumors - which are a result of the disease spreading to other parts of the body - had yet more different genetic faults, suggesting that basing treatment decisions on just one primary tumor sample is not sufficient.

"We've known for some time that tumors are a patchwork of faults, but this is the first time we've been able to use cutting-edge genome sequencing technology to map out the genetic landscape of a tumor in such exquisite detail," said Charles Swanton, of University College London's cancer institute, who led the study and presented its results at a briefing in London on Tuesday.

He said they had uncovered "an extraordinary amount of diversity" at a genetic level both within tumors and within a single patient, with more differences between biopsies from the same tumor than similarities.

"The next step will be to understand what's driving this diversity in different cancers and identify key driver mutations that are common throughout all parts of a tumor," Swanton said.

PERSONALISED MEDICINE

Genetic profiling of patients and their tumors has become more common in cancer treatment in wealthy countries as drug companies develop new generations of so-called "personalized medicines" that target cancers with specific genetic features.

Roche's blockbuster breast cancer drug Herceptin is designed to treat only women who make too much of the HER2 protein, for example, while Novartis's Afinitor targets mTOR, a protein that acts as an important regulator of tumor cell division, blood vessel growth and cell metabolism.

James Larkin, an oncologist at London's Royal Marsden Hospital who also worked on the study, said the findings suggest the reality of personalized cancer treatment is far more complex than previously thought.

"The molecular changes that drive the growth of the cancer once it has spread may be different from those that drive the growth of the primary tumor," he said.

The researchers compared genetic faults in various tumor samples taken from the four patients.

They found 118 different mutations - 40 of which were "ubiquitous mutations" found in all biopsies, 53 "shared mutations" that were found in most but not all biopsies, and 25 "private mutations" only found in a single sample.

By analyzing where the shared mutations were in relation to the whole tumor, the researchers were able to trace the origins of certain subtypes of cancer cells back to what they called key "driver mutations." This allowed them to create a map of how the pattern of faults might have evolved over time.

Swanton likened the findings to a tree, in which the trunk is the primary tumor and the branches the secondary tumors from the cancer's spread.

While he stressed the results would need to be replicated with larger numbers of patients and in different types of cancer, he said these early indications showed "the importance of targeting common mutations found in the trunk of the tree as opposed to those found in the branches."

"It may also explain why surgery to remove the primary kidney tumor can improve survival," he added, since cutting out a tumor reduces the risk that cells resistant to drug treatment could go on to re-grow the tumor or spread elsewhere.

source: mb.com.ph